SUU, Alm.del - 2019-20 - Bilag 446: Henvendelse af 23/6-20 fra Bo Hembæk Svensson om sammenhæng mellem toxoplasmose og covid-19

Sundheds- og Ældreudvalget 2019-20
SUU Alm.del Bilag 446
Offentligt

Copenhagen, June 23rd, 2020.

v.1.0

Bo Hembæk Svensson,

Toxoplasma Research

[email protected]

To: Sundhedsministeriet, Sundhedsstyrelsen og Statens Serum Institut

cc: Arbejdsgruppen for Tg v/FVST, Folketingets Sundhedsudvalg

cc:

Whom it may concern.

Note IV: Further Evidence that Covid-19 provokes the onset of acute toxoplasmosis. Toxoplasma has

always been grossly underreported. Resistance among authorities towards examining the correlation.

As documented earlier (here,

here

and

here)

there is a strong correlation between Covid-19 and

toxoplasmosis as there are obvious and scientifically documented overlaps in symptoms, outcomes,

treatments, and age distribution - rendering Covid-19 and Toxoplasmosis closely correlated. Yet there is no

protocol in place anywhere in the world that tests Covid-19 patients for toxoplasmosis.

Further evidence has emerged in May and June, adding weight to the strong correlation between Covid-19

and Toxoplasma infection, e.g toxoplasma induced microvascular dysfunction/damage, toxoplasma induced

hypoxia and the fact that the latest drug Dexamethasone is effective towards toxoplasmosis too.

A. Every normal and “abnormal” Corona/Covid-19 symptom reported, is virtually always mirrored in

known symptoms in acute toxoplasmosis.

It is well known that “viruses kill bacteria”, and at least that there are many interactions between

different pathogens,

“

Infection against infection: parasite antagonism against parasites, viruses

and bacteria

”.

Some 95% of the lethal Covid-19 cases are related to “secondary infections” - and obviously a

number of these have a high probability of being caused by acute toxoplasmosis.

SUU, Alm.del - 2019-20 - Bilag 446: Henvendelse af 23/6-20 fra Bo Hembæk Svensson om sammenhæng mellem toxoplasmose og covid-19

B. Tg is extremely underreported and poorly understood among health workers. Since it was declared

"latent & benign" some 50 years ago, it has been virtually eliminated from the curriculum. Only few

doctors know the multiple symptoms of acute- or latent toxoplasmosis, and even fewer have even

diagnosed it.

It is remarkable that 100% of patients that were checked/positive for lung-cancer

also had acute toxoplasmosis (i.e parasites in the bloodstream)

- but with no diagnosis.

“Toxoplasmosis has the highest human incidence amongst the parasitic zoonoses.

However, toxoplasmosis is considered to be an under-detected and underreported

disease in the European Union

”

C. Every single suggested treatment/drug etc. for Covid-19, is mirrored in being effective against

Toxoplasma too. This also goes for the latest discovery regarding Dexamethasone being effective in

Covid-treatment:

a. (Covid) "WHO welcomes preliminary results about dexamethasone use in treating critically

ill COVID-19 patients"

b. (Toxoplasma) "Conclusion. IVCD (i.e dexamethasone) is an effective route of treatment for

active toxoplasmic retinochoroiditis..." and “A large study is ongoing to find the right

dosage of dexamethasone: "Cerebral toxoplasmosis is the most common opportunistic

infection in HIV patients resulting in up to 50% of mortality with proper treatment and 80%

without it."

D. Also, it has emerged that CD4 and CD8 T-cells play critical roles in both pathologies and therefore

easily can interrelate/interfere both ways, e.g:

a. (Covid) : Collectively, CD4+ T-cells from severe COVID-19 patients are hyperactivated and

FOXP3-mediated negative feedback mechanisms are impaired in the lung, while activated

CD4+ T-cells continue to promote further viral infection through the production of Furin.

Therefore, our study proposes a new model of T-cell hyperactivation and paralysis that

drives pulmonary damage, systemic CRS and organ failure in severe COVID-19 patients.

b. (Toxoplasma): Although CD8 T cells play an important effector role in controlling the

chronic (Tg) infection, their maintenance is dependent on the critical help provided by CD4

T cells. In a recent study, we demonstrated that reactivation of the infection in chronically

infected host is a consequence of CD8 T dysfunction caused by CD4 T cell exhaustion.

SUU, Alm.del - 2019-20 - Bilag 446: Henvendelse af 23/6-20 fra Bo Hembæk Svensson om sammenhæng mellem toxoplasmose og covid-19

The danish health authority at SSI acknowledges the general correlation between Toxoplasma and

Covid-19, but has in May 2020

declined to examine it further with the argument that there is ”no scientific

evidence”, which we have to dispute.

We argue that there is much evidence connecting the two diseases, and that the totality of this evidence

warrants a thorough examination of the hypothesis that Covic-19 is a Toxoplasma antagonist

- and we

have offered all the knowledge and information we have to support this.

The Toxoplasma parasites psychological hallmark is to “manage fear” - and if anything is driving the

current developments, it is fear.

We strongly recommend that the danish health authorities take this proposition seriously, and not only

recognizes the correlations but also implements toxoplasma testing Covid-19 patients and of tissue- and

blood samples. We believe that active, acute toxoplasmosis will be identified in +50% of the cases.

We will, of course, share all documents and findings with anyone inclined to help elucidate the possible

Covid-19/Toxoplasma correlation.

All citations above are form peer-reviewed scientific articles from recognized journals.